Author
Information
Poonia S*, Satia MN**, Torame VP***, Natraj G****.
(* Third
Year Resident, ** Prodessor, Department of Obstetrics and
Gynecology; *** Assistant Professor, **** Professor, Department of Microbiology; Seth GS Medical College
& KEM Hospital, Mumbai, India.)
Abstract
A case of typhoid fever
in second trimester of pregnancy with vertical transmission leading to
intrauterine fetal death at 14 weeks of gestation has generated curiosity about
organisms that cross placenta in pregnancy. Salmonella typhi was isolated in the
retained products of conception which were sent for culture and sensitivity as
patient presented with symptoms and signs of pelvic infection.
Introduction
Typhoid fever is a major
health problem in developing countries. Cases of vertical transmission have
been reported from India.[1][2]Pregnant women are susceptible to
food borne infections like typhoid owing
to hormonal changes that suppress immunity.[3]Salmonella typhi can cross the
placenta and cause miscarriage, still births and preterm labor. We report a
case where a Para 1 Living 1 with history of
fever and inevitable abortion grew Salmonella typhi on her retained
products of conception.
Case Report
A 22 year old Gravida 2
Para 1 Living 1 woman with 14 weeks of gestation
came with pain in abdomen and spotting per vaginum since one day.
She had
history of fever off and on one week back, for which she had taken antibiotics
from a private practitioner, the details of
which were not
available. She had registered at 6 weeks of gestation and pregnancy was
confirmed by ultrasonography. She had a fever spike of 380 C after admission. Her
vital parameters were stable. On
abdominal examination, her uterus was corresponding to 14 weeks of gestation. Bimanual
pelvic
examination showed the uterus to be corresponding to 14 weeks, cervical os was
open and products of conception felt were through it. Vagina was warm and
uterus was tender. Based on history, clinical findings, and geographical
endemicity of acute onset fever pathogens, her blood samples were sent. Her
hemoglobin was 9.4 g/dL, white blood cell count 7300/cmm, and platelets of 0.66/cmm. Her urine culture
was sterile. Widal titre was positive up to dilution of 1: 240 for both Salmonella
typhi
‘O’ (somatic) and Salmonella typhi’ H’ (flagella). Rapid test for malaria,
dengue and leptospirosis were negative. Liver and renal function parameters
were within normal limits. Blood sugars and thyroid profile were normal. A curettage was done for
inevitable and septic abortion under antibiotic cover
(ceftriaxone and metronidazole). Products of conception were sent for culture
and sensitivity. Salmonella typhi was isolated from products of conception
and further confirmed by Salmonella polyvalent antisera.
Salmonella typhi was sensitive to
ceftriaxone, carbapenems, other third generation cephalosporins, cotrimoxazole
and chloramphenicol. The isolate was a nalidixic acid resistant Salmonella
Typhi (NARST) which was also resistant to ciprofloxacin. Her blood culture and stool culture sent on day 3 of admission,
did not reveal any Salmonella growth. She was
treated with same antibiotics for 5 days and was discharged on day 5 of
procedure.
In the absence of any
other likely cause of spontaneous miscarriage in this case, Salmonella typhi
was presumed to be the most likely cause.
Discussion
Incidence of infection
with Salmonella in pregnant patients is similar to general population (0.2%).[4]
Typhoid
is caused by Salmonella enterica
serotype typhi. The most common route
of transmission is feco-oral route. In humans young, old, pregnant, HIV
infected and patients who have undergone transplant are at higher risk for
Salmonella infection.[5] Vertical transmission of
Salmonella occurs via transplacental spread or because of the bacteraemia
during labor or due to inadvertent fecal contamination of birth canal.
Incidence of fetal loss in untreated typhoid can be as high as 80%.[4]
Salmonella
typhi
has been associated with abortions in animals like sheep, cattle and horses.[6]
Salmonella typhi is an intracellular
bacterium that resides within the modified phagosomes of Antigen Presenting
cells (APCs). Innate immunity is important in curtailing infection during
the first
week of infection and CD4 T-cells response to Salmonella typhi is detectable only
after 7 days of infection whereas CD8 T-cells response is delayed until second
week post infection.[7] Overall, Salmonella typhi has evolved many
mechanisms to evade the host immune system. In pregnancy, there is a shift in
immune status from type 1 (cell mediated immunity) to type 2 (humoral immunity).This
shift in immune system though is more pronounced at the maternal fetal
interface and may also affect systemic immunity.[8]
Various organism other
than TORCH (Toxoplasma gondii, Rubella virus, Cytomegalovirus, Herpes Simplex
virus) that infect human placenta have been reported in literature.[9]
These
organisms are intracellular for a portion of their lifetime and
infect placenta via hematogenous spread. Salmonella being an intracellular
organism are recruited at the fetal implantation site early in illness before
treatment or during an episode of bacteremia. The extravillous trophoblast with
immune modifications are juxtaposed near these maternal decidual cells. Due to
various invasive and evasive strategies Salmonella typhi may cause
significant damage to mother and fetus.
As per reports by Hick’s
and French, Salmonella typhi may cross placenta and cause miscarriage (65-80%),
stillbirths and preterm labour.[10]
Transplacental
transmission usually presents as spontaneous second trimester abortion without
premature rupture of membranes. Neonates born to mother with Salmonellosis are
more prone to severe complications like septicaemia and meningitis.[11]
Ampicillin or
amoxicillin are considered as first line drugs during pregnancy. Ceftriaxone is
the preferred drug in nalidixic acid resistant Salmonella typhi (NARST), also
resistant to ciprofloxacin.[12] Typhoid vaccines (both polysaccharide and
live vaccine) are category C drugs during pregnancy.
Salmonellosis is usually
not included in the differential diagnosis of miscarriage, stillbirth or
neonatal sepsis that occur during pregnancy. However it should be considered to
prevent fetal and mother morbidity.
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Poonia S, Satia MN, Torame VP, Natraj G. Vertical transmission of Salmonella typhi. JPGO
2015. Volume 2 No. 1. Available from: http://www.jpgo.org/2015/01/vertical-transmission-of-salmonella.html